Can persistent hyperglycemia and insulin resistance in type 2 diabetics be due to decreased transcapillary movements of insulin (resulted from cell swellings in blood and in vascular walls) by hypotonicity of blood due to elevated glucose or otherwise? Since the insulin molecule is bigger, does it result in decreased insulin exposure to target cells instead of commonly thought decreased sensitiveness of insulin? This looks quite relevant and simple reasoning to insulin resistance or persisting hyperglycemia.

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